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Methods In 1996 and 1997, we measured thyrotropin in stored serum samples collected from 25,216 pregnant women between January 1987 and March 1990. We then located 47 women with serum thyrotropin concentrations at or above the 99.7th percentile of the values for all the pregnant women, 15 women with values between the 98th and 99.6th percentiles, inclusive, in combination with low thyroxine levels, and 124 matched women with normal values.

Their seven-to-nine-year-old children, none of whom had hypothyroidism as newborns, underwent 15 tests relating to intelligence, attention, language, reading ability, school performance, and visual–motor performance. Results The children of the 62 women with high serum thyrotropin concentrations performed slightly less well on all 15 tests. Their full-scale IQ scores on the Wechsler Intelligence Scale for Children, third edition, averaged 4 points lower than those of the children of the 124 matched control women (P=0.06); 15 percent had scores of 85 or less, as compared with 5 percent of the matched control children.

Of the 62 women with thyroid deficiency, 48 were not treated for the condition during the pregnancy under study. The full-scale IQ scores of their children averaged 7 points lower than those of the 124 matched control children (P=0.005); 19 percent had scores of 85 or less. Eleven years after the pregnancy under study, 64 percent of the untreated women and 4 percent of the matched control women had confirmed hypothyroidism. The link between hypothyroidism caused by iodine deficiency during pregnancy and mental retardation in the offspring has been recognized for nearly 100 years. Iodine deficiency is associated with thyroid deficiency in both mother and fetus, a situation that makes it impossible to determine whether the mental retardation of the fetus is due to maternal hypothyroidism or both maternal and fetal hypothyroidism. In developed countries, chronic autoimmune thyroiditis is the most common cause of hypothyroidism among women in their childbearing years. Antibodies responsible for compromising maternal thyroid function can cross the placenta and, in some instances, compromise fetal and neonatal thyroid function.

One such antibody, the thyrotropin-receptor–blocking antibody, has been implicated in cases of transient congenital hypothyroidism that were identified by screening programs for newborns. In 1969, Man and Jones suggested that mild maternal hypothyroidism alone was associated with lower IQ levels in the offspring; their study involved a cohort of 1349 children, and measurements of serum butanol extractable iodine were used to distinguish between euthyroidism and hypothyroidism in women. A study by Matsuura and Konishi in 1990 documented that fetal brain development is adversely affected when both the mother and fetus have hypothyroidism caused by chronic autoimmune thyroiditis. In such cases, transient neonatal hypothyroidism is present. In an earlier, population-based survey of 2000 pregnancies, we measured serum thyrotropin concentrations during the second trimester. The concentrations were high (above 6 mU per liter) in 49 of the women, of whom 6 (3 per 1000) had low serum free thyroxine concentrations. If a lowering of the IQ levels of the offspring were to occur sufficiently often in association with this degree and frequency of maternal thyroid deficiency, then systematically determining the thyroid status of all women before or very early in pregnancy might be justified.

The aim of the current study was to determine whether undetected or inadequately treated maternal thyroid deficiency during pregnancy is associated with lower IQ scores in the offspring in the absence of neonatal hypothyroidism. Methods The Foundation for Blood Research administers a statewide, second-trimester prenatal serum screening program for open neural-tube defects and Down's syndrome in Maine. Aliquots of serum that remain after screening are routinely coded and stored at –20°C. Outcome information is available through a contract with the state's Bureau of Vital Records.

The current study cohort was limited to women with viable singleton pregnancies, who were screened between January 1987 and March 1990, and their infants whose birth weight was at least 1500 g. The serum from the mothers was shipped to the New England Newborn Screening Program in Boston, where serum thyrotropin was measured. Samples from 25,216 women were analyzed in five batches over a two-year period. Selection of Women with Hypothyroidism and Control Subjects We recruited women with hypothyroidism during pregnancy, as determined by a high serum thyrotropin concentration, without regard to treatment status, and we tested their children between March 1996 and December 1997.

We contacted 55 of the 75 pregnant women with serum thyrotropin concentrations at or above the 99.7th percentile of the values for all the pregnant women; 47 (85 percent) agreed to participate. In 2 of the 75 pregnancies, the women were enrolled through a previous pregnancy. Of the 18 women not contacted, 3 had moved to another state, 1 had died, and for 1, the child was a ward of the state.

The remaining 13 were not contacted because of limited funds. At the urging of a grant review panel, we recruited 18 more women to represent a range of milder cases, defined by a serum thyrotropin concentration between the 98th and 99.6th percentiles, inclusive, and a serum thyroxine concentration below 7.75 μg per deciliter (99.7 nmol per liter). To identify this second subgroup, we measured serum thyroxine concentrations in 247 pregnant women with serum thyrotropin concentrations between the 98th and 99.7th percentiles. Fifteen of the 18 women identified (83 percent) agreed to participate. After recruitment, we measured thyroxine, free thyroxine, and antithyroid peroxidase antibodies in the original serum samples from all women in the study. For each woman with hypothyroidism, we identified potential control subjects who had serum thyrotropin concentrations below the 98th percentile and who were matched according to the following criteria: mother's age at delivery (within one year), number of years of education of the mother (within one year), gestational age at the time of sampling (same completed week), duration of storage of serum sample (within one month), and sex of the child.

From this group, two women were randomly selected and recruited for each woman with hypothyroidism. Additional matched control women were available from the same list, if one initially declined participation.

The protocols for the additional assays and the follow-up study were approved by the institutional review board at the Foundation for Blood Research. Enrollment began with a telephone call to the woman and a letter describing the study. Then an appointment was arranged, at which informed consent was requested and, if consent was granted, testing was performed on the child. The neuropsychological test results were provided to the family within one month after the child's testing was completed. At the end of the study, we contacted the women with hypothyroidism and the matched control women again to determine whether hypothyroidism had been clinically diagnosed since the pregnancy in those who had not received a diagnosis of hypothyroidism at the time of pregnancy.

The contact was initially by a letter, which also included information about the thyrotropin concentrations in the stored serum samples. The letter was followed by a telephone call, during which a questionnaire was administered and a blood test for measuring thyrotropin was offered. For those who agreed to be tested, blood spots were collected on filter paper by a finger prick. Study Procedures We collected standardized information about socioeconomic status and medical history from all women enrolled in the study, using the Four Factor Index of Social Status (the Hollingshead score). Each woman and her husband or partner were assigned an education code ranging from 1 (corresponding to less than seven years of schooling) to 7 (corresponding to graduate or professional training) and an occupation code ranging from 1 (e.g., “farm worker”) to 9 (e.g., “higher executive”). Each couple's individual education scores were multiplied by 3, the occupation scores were multiplied by 5, and the two values were then added together. The final score was the average of the scores of the woman and her partner.

When one partner was not employed, the final score was taken to be the score of the employed partner. The woman was also asked whether her child had repeated a grade and about her child's school performance, including whether the child had had learning problems or other difficulties in school. Neuropsychological testing of the women's children included assessment of intelligence, attention, language, reading ability, school performance, and visual–motor performance. One of two certified psychologists performed the testing, and the project's consulting psychologist supervised the testing and rescored the tests. The staff involved in the neuropsychological testing did not know whether the children's mothers were women with hypothyroidism or control women. Intelligence was measured with use of the Wechsler Intelligence Scale for Children, third edition, the most widely used intelligence test.

This test provides a full-scale IQ score and subscale scores (range, 40 to 160) for verbal skills, performance, and freedom from distractibility. To measure hearing deficits in the children, the staff administered subtests on word discrimination and word articulation from the Test of Language Development, second edition (range of scores, 1 to 20). We used the norms for children 8 years 11 months of age, because the version for older children did not have scales for word discrimination or articulation. The Peabody Individual Achievement Test, revised (PIAT-R), was used to measure reading recognition and reading comprehension (range of scores, 55 to 145). The staff administered the Conners' Continuous Performance Test to measure sustained vigilance and attention, using a computer program that employs a go–no go paradigm (range of overall index score, 1 to 30).

The Developmental Test of Visual-Motor Integration was administered to provide a standard measure of visual perception and fine motor skills (range, 55 to 145). The grooved pegboard test was administered to assess visual–motor coordination and dexterity by measuring the time required to insert pegs with both the preferred and nonpreferred hand (for this test, it is recommended that normative data be derived from control children in the study). Assay Methods We measured serum thyrotropin using a coated-tube radioimmunoassay (Diagnostic Products, Los Angeles).

Thyrotropin was measured on dried blood spots with a time-resolved immunofluorometric assay (Wallac Oy, Turku, Finland). Serum thyroxine was measured with a solid-phase radioimmunoassay or a time-resolved immunofluorometric assay (Wallac Oy); serum free thyroxine was measured with a time-resolved immunofluorometric assay. We measured serum antithyroid peroxidase antibodies using the Kalibre enzyme-linked immunosorbent assay (Kronus, San Clemente, Calif.) (normal concentration, ≤2 U per milliliter). Statistical Analysis The serum thyrotropin, thyroxine, and free thyroxine concentrations were logarithmically transformed before analysis. We used geometric means and logarithmic standard deviations to summarize the results (after censoring seven measurements that were more than 3 SD above or below the group mean).

We compared categorical variables using an exact test of significance or odds ratios, and we compared continuous variables using the Student's t-test. When necessary, the t-test was modified to allow for unequal variances. The primary analysis was of all 62 women with hypothyroidism and all 124 control women; we preserved matching by comparing the result from the child of a woman with hypothyroidism with the average result from the two matched control children.

No adjustment was made for multiple comparisons. All statistical tests were two-sided.

Results According to records from the New England Newborn Screening Program, none of the children of the 62 women with high serum thyrotropin concentrations while pregnant were identified as having transient or permanent congenital hypothyroidism. The distribution of serum thyrotropin concentrations in the 62 women with hypothyroidism and the 124 matched control women is shown in Figure 1 Distribution of Serum Thyrotropin Concentrations during Pregnancy in the 62 Women with Hypothyroidism and the 124 Matched Control Women. Open circles indicate the 14 women who were treated for hypothyroidism during the pregnancy under study. Selected percentiles are shown for the entire cohort of 25,216 pregnant women..

Fifteen of the 62 women with hypothyroidism reported that they had received a diagnosis of hypothyroidism before the pregnancy, and 14 of these 15 women were treated for hypothyroidism during that pregnancy. Two of the control women reported that they had had hypothyroidism in the distant past but were never treated. Demographic and pregnancy-related information about the women with hypothyroidism and the control women and the remainder of the cohort from which the women were selected is shown in Table 1 Characteristics of the Study Women, the Remainder of the Cohort, and Their Children.. There were no significant differences between the women with hypothyroidism and the control women for any of the variables. Four of the variables were used for matching: number of years of education of the mother, mother's age at delivery, gestational week when the serum sample was obtained, and sex of the child.

The use of the number of years of education as a matching variable was intended to control for socioeconomic status. To assess the effectiveness of this matching, we used the Hollingshead score as an additional measure. This score took into account the mother's educational level and occupation and also the father's educational level and occupation.

The mean Hollingshead score in the women with hypothyroidism was one point lower than that in the control women (P=0.43). The study children were similar in most respects to the remainder of the cohort, but more were girls, their mothers were older, a higher percentage of their mothers were married, and more of their mothers were multiparous.

The results of the measurements of thyroid function in the women with hypothyroidism and the control women during pregnancy are shown in Table 2 Measurements of Thyroid Function in the Study Women during Pregnancy.. As expected, according to the selection process, the women with hypothyroidism had higher serum thyrotropin and lower serum thyroxine concentrations. The children's neuropsychological test scores are shown in Table 3 Neuropsychological Test Scores among the Children of Women with Hypothyroidism during Pregnancy as Compared with the Children of Matched Control Women.. All the children were between seven and nine years of age when tested, and the child of a woman with hypothyroidism and his or her matched control children were tested at the same age.

The analysis preserved matching for each of the tests by expressing the relative performance between case and control children as a mean difference (the value in the case child minus the average of the values in the two control children). The case children performed less well on all the tests; 2 of the 15 differences reached statistical significance (P. Discussion The current study shows that hypothyroidism in pregnant women can adversely affect their children's subsequent performance on neuropsychological tests. Decreases in performance can occur even when the pregnant woman's hypothyroidism is mild and probably asymptomatic.

The presence of high serum concentrations of antithyroid peroxidase antibodies in 77 percent of the women with hypothyroidism indicates that chronic autoimmune thyroiditis was the most frequent cause of hypothyroidism in these women. Treating maternal hypothyroidism during pregnancy appears to be beneficial for the child, even when treatment is inadequate as determined by measurements of thyrotropin. If our findings were to be confirmed, and routine screening for hypothyroidism during pregnancy were to be instituted, what might the benefits be? The main benefit — an increase of approximately 4 points in IQ scores — would occur in the children of women with serum thyrotropin concentrations at or above the 98th percentile. A secondary benefit would be reduced morbidity for women who were systematically identified and treated. The present study shows that a large percentage of pregnant women with high serum thyrotropin concentrations subsequently have clinically apparent hypothyroidism. Because the symptoms associated with hypothyroidism are nonspecific, the condition can be difficult to diagnose, as reflected by the five-year median time to diagnosis in the women.

Before about 12 weeks' gestation, when the fetal thyroid gland becomes active, the mother is the sole source of thyroid hormones. Maternal thyroid sufficiency might therefore be most important in the first trimester.

This theory is supported by a recent study in a small cohort of 220 healthy infants in which lower maternal serum free thyroxine concentrations at 12 weeks' gestation were associated with impaired psychomotor development at 10 months of age. However, the later stages of fetal brain development involve neuronal migration and organization. Since these processes are responsible for functions measured by the neuropsychological tests used in the present study, thyroid insufficiency beyond the first trimester is also likely to have adverse effects. In rats, triiodothyronine receptors are first detected in the brain in the second trimester, and the induction by triiodothyronine of enzymes that are important in nervous-system development begins late in fetal development. The current study documents a relatively long average interval between early biochemical evidence of hypothyroidism and clinical diagnosis, a finding that suggests that ongoing maternal health problems might hinder the child's development after birth. In the absence of objective data, the most prudent policy would be to identify and treat maternal hypothyroidism as early in pregnancy as possible, keeping in mind that the need for thyroxine increases during pregnancy.

We conclude that systematic screening for hypothyroidism early in pregnancy may be worthwhile, even when the degree of deficiency is mild and does not cause immediate clinical manifestations in the woman. If routine screening were to be introduced, the most conservative policy would be to perform testing at the first prenatal visit, preferably in the first trimester. Follow-up of women with positive screening results would need to be prompt, so that treatment could begin quickly. References • 1 Gardner LI. Historical notes on cretinism. In: Gardner LI, ed. Endocrine and genetic diseases of childhood and adolescence.

Philadelphia: W.B. Saunders, 1975:234-8. • 2 Pharoah POD, Connolly KJ, Ekins RP, Harding AG. Maternal thyroid hormone levels in pregnancy and the subsequent cognitive and motor performance of the children. Clin Endocrinol (Oxf) 1984;21:265-270 • 3 Sutherland JM, Esselborn VM, Burket RL, Skillman TB, Benson JT. Familial nongoitrous cretinism apparently due to maternal antithyroid antibody: report of a family.

N Engl J Med 1960;263:336-341 • 4 Connors HM, Styne DM. Transient neonatal “athyreosis“ resulting from thyrotropin-binding inhibitory immunoglobulins. Pediatrics 1986;78:287-290 • 5 Cho BY, Shong YK, Lee HK, Koh CS, Min HK, Lee M. Transient neonatal hypothyroidism due to transplacental transfer of maternal immunoglobulins that inhibit TSH binding, TSH-induced cAMP increase and cell growth. Endocrinol Jpn 1988;35:819-826 • 6 Fort P, Lifshitz F, Pugliese M, Klein I. Neonatal thyroid disease: differential expression in three successive offspring.

J Clin Endocrinol Metab 1988;66:645-647 • 7 Goldsmith RE, McAdams AJ, Larsen PR, McKenzie M, Hess EV. Familial autoimmune thyroiditis: maternal-fetal relationship and the role of generalized autoimmunity. J Clin Endocrinol Metab 1973;37:265-275 • 8 Man EB, Jones WS. Thyroid function in human pregnancy.

Incidence of maternal serum low butanol-extractable iodines and of normal gestational TBG and TBPA capacities: retardation of 8-month-old infants. Am J Obstet Gynecol 1969;104:898-908 • 9 Dussault JH, Fisher DA. Thyroid function in mothers of hypothyroid newborns. Obstet Gynecol 1999;93:15-20 • 10 Matsuura N, Konishi J. Transient hypothyroidism in infants born to mothers with chronic thyroiditis -- a nationwide study of twenty-three cases.

Endocrinol Jpn 1990;37:369-379[Erratum, Endocrinol Jpn 1990;37:767a.] • 11 Klein RZ, Haddow JE, Faix JD, et al. Prevalence of thyroid deficiency in pregnant women.

Clin Endocrinol (Oxf) 1991;35:41-46 • 12 Haddow JE, Kloza EM, Smith DE, Knight GJ. Data from an alpha-fetoprotein pilot screening program in Maine. Obstet Gynecol 1983;62:556-560 • 13 Haddow JE, Palomaki GE, Knight GJ, et al. Prenatal screening for Down's syndrome with use of maternal serum markers. N Engl J Med 1992;327:588-593 • 14 Hollingshead AB. Four factor index of social status.

New Haven, Conn.: Yale University, 1975. • 15 Wechsler D.

Wechsler intelligence scale for children. San Antonio, Tex.: Psychological, 1991. • 16 Newcomer P, Hammill D. Test of language development — 2: intermediate. Austin, Tex.: PRO-ED, 1991. • 17 Markwardt FC.

Peabody individual achievement test. Circle Pines, N.M.: American Guidance Service, 1989. • 18 Conners CK. Conners' CPT computer program. North Tonawanda, N.Y.: Multi-Health Systems, 1994. (software & user's manual). • 19 Beery KE.

Developmental test of visual-motor integration. Cleveland: Modern Curriculum Press, 1989. • 20 Trites R.

Grooved pegboard test. Lafayette, Ind.: Lafayette Instrument, 1989. • 21 Mitchell ML. Improved thyroxine radioimmunoassay, for filter paper discs saturated with dried blood. Clin Chem 19-1914 • 22 Pop VJ, Kuijpens JL, van Baar AL, et al. Low maternal free thyroxine concentrations during early pregnancy are associated with impaired psychomotor development in infancy. Clin Endocrinol (Oxf) 1999;50:149-155 • 23 Neuronal proliferation, migration, organization and myelination.

In: Volpe JJ. Neurology of the newborn. Philadelphia: W.B. Saunders, 1995:43-92. • 24 Oppenheimer JH, Schwartz HL. Molecular basis of thyroid hormone-dependent brain development.

Endocr Rev 1997;18:462-475 • 25 Mandel SJ, Larsen PR, Seely EW, Brent GA. Increased need for thyroxine during pregnancy in women with primary hypothyroidism. N Engl J Med 1990;323:91-96 .

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This is a Tactics Ogre: Let Us Cling Together FAQ and guide focused on the PSP remake. The objective of the guide is to give the player a basic understanding of the game as well as cover the main scenario of the game. Therefore, it covers all the routes in the main scenario and how to recruit Jun 18, 2011 Here is the link: For most of the equipment that is dropped by enemies, I have given you enough information to get to the right part of a dungeon and know which enemy you're looking for. You can find them A website dedicated to Tactics Ogre: The Knight of Lodis and Tactics Ogre: Let Us Cling Together.

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A typical command would be like this below: Code: [Select]. & sex -c parameter.sex 1 Feb 2017 4.

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I was watching the transporter and he had a 7 series that is manual. I know a lof of crap is fake in that movie. But the 7 series got me boggled.

24 Nov 2013 30 Jul 2008 Post Count: 1,571. Likes Received:1. In the movie, the car is said to be a 1999 735i.

However, I read in other online sites that the car was a V-12 (making it a 750) converted to have the manual trans. Does anyone know which is correct? Also, did any E38's come with a manual gearbox? (Euro models). A page that describes the BMW vehicle used in The Transporter movie.

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30 Jan 2013 1: The Transporter – 735i. We said “in no particular order”, but this one may be our favorite.

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Your rating: Not rated - login - and Feb 13, 2011 Apparently, I shouldn't consign some of those great old DOS games of yore to the round file just yet. Try DOSBox, an opensource program that emulated DOS on faster operating systems.

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Secret agent man #1, 1990 Joined: 5/3/2007. From: east coast, usa. Status: online, DFend has moved on to 'DOSBox Game Launcher'. If you have trouble with other DosBoxes, you may want to try this one Running the game with this start.bat file automaticaly adjusts the cycles to 60000 which seems to be good for Steel Panthers 2. Mar 14, 2014 Originally Posted by NGTM1R View Post. This suggestion is not possible, as the game download does not appear to include any executable file that dosbox can run.

'intro' is a Windows NT executable, 'Mech' is a PE executable, whatever that means.). Feb 21, 2013 Aug 11, 2014,,,,. Download Fortresscraft evolved lift manual control valve: Read Online Fortresscraft evolved lift manual control valve: Dec 20, 2015 Okay, just repoduced the getting on lift at bottom of range with manual control module resulting in your falling through lift. Investigating now. That came back again for meit's very odd, seems once its starts happening it will keep happing but can go away if you reload. Also once that problem at the bottom Aug 23, 2015 pull myself back to the ground!

It's kind of cool but probably NOT intented.:) The above reverse gravity bit seems to happen when destroying a lift while ON a lift if I recall correctly. The random 'hey you're dead!' While riding a lift up well that seems to be random. It was on a manual control lift if that helps. Mar 31, 2014 Lifts - numbers and stuff.

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Falling through the lift, lift not rising to my level, etc. The one thing I did that solved the problem, was to remove the manual control module. Alternative to manual control? Well, my shaft has working spaces carved out Jul 26, 2014 I'm afraid so; 64 is a technical limitation right now. The manual control is supposed to be expensive, and popped onto a lift when you need it to be useful (I often use it to populate shafts of conveyors, for instance) Eventually, it's likely that the lift control module will just be something you can carry that allows Controls the lift. It must be installed adjacent to the lift block. If it is not touching a lift Feb 22, 2017 Feb 22 @ 8:35am.

You'll adjust height by looking at the base of the lift itself, it will highlight Q and E I believe for adjustment. After a compressor is attached and powered, by default, it will still lift to 64 until you adjust it. You can add a remote for manual control.

Last edited by DustBust; Feb 22 @ 8:36am. Aug 28, 2015 Could be solved with a sensor block and some kind of redstone/laser to connect it to the elevator control. Stand on the block and the elevator gets called to that exact location, regardeless of max blocks. This way you could also have different levels where you can call the elevator to, different mining shafts,,,,. Download Nz group guidelines and rules: Read Online Nz group guidelines and rules: social club rules template work social club rules nz society rules for members rules of society examples meeting procedures and rules nz rules and regulation of an association annual general meeting rules nz incorporated society rules template Group work guidelines for students. Group activities. Middle stages.

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Attach to Form 1120, 1120-C, 1120-F, 1120S, 1065, or 1065-B.? Information about Form 1125-A and its instructions is. Section references are to the Internal Revenue Code unless otherwise noted. Use Form 1125-A to calculate and deduct cost of goods sold for certain entities. This form is used by individuals to request their own driving record or their vehicle/vessel information. A $5 fee is required for each record requested. If you want your driving record or vehicle/vessel record certified as a true copy on file with DMV; write on the top of the INF 1125 form that you would like to request a 'certified Internal Revenue Service.

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Instructions for Form 1125-E. (December 2011). Compensation of Officers. Section references are to the Internal Revenue Code unless.

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Certain entities with total receipts of $500000 or more use this form to provide a detailed report of the deduction for compensation of officers. Form 1125-E must be completed and attached to Form 1120, 1120-C, 1120-F, 1120-RIC, 1120-REIT, or 1120S, if the entity has total receipts (defined below) of $500,000 or more, and deducts compensation for officers. For purposes of Form 1125-E, total receipts are determined as follows.://clashroyaledeckbuilder.com/viewDeck/,,,,. Download Everquest iksar monk leveling guide: Read Online Everquest iksar monk leveling guide: p99 monk solo guide iksar leveling guide project 1999 p1999 monk solo guide project 1999 iksar shaman guide eq monk solo guide p99 sk solo everquest iksar necromancer guide p99 monk guide 28 Apr 2011 I leveled up in Kunark as an iksar monk. I didn't really do much of a grind-as-fast-as-i-can thing back then but i leveled solo in OT until i could get a ledge group there where i solo/group until i could get a group in Dreadlands where i did the same stuff until i could get groups in Karnors Castle (my all time 24 Mar 2016 I am currently a L 10 Iksar monk on Phinney.

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My early notes about soloing an Iksar necromancer and wood elf druid in old-school Everquest 1. Mostly for you nostalgic Everquest 1 progression or Project 1999 solo guide A skeleton with a shield means 1pp from vendoring the shield, which is two backpacks from just one level 1 mob that doesn't run. Obvs, don't Hello everyone, I'm making this thread in hopes of having a couple questions answered and to create a resource for people looking to level a monk.

It seems like the community is lacking in a decent guide for leveling monks so I thought this might help. I'm rolling with a 42 Iksar (Master Race) monk, he's in 20 Apr 2016 No matter if it's the Plane of Time, level 70 experience groups or raid pulling - Sampson has really seen a lot of EverQuest, analysed dozens of end-level encounters, fought in some of the most challenging zone and soloed for a little fortune. In his EverQuest monk guide he now shares his knowledge with you: [Archive] Monk SOLO Guide Server Chat. Any super knowledgeable Iksar Monks out there that have the primo solo guide for Iksar Monks?

Doesnt have to be only Kunark spots. My guide would start.

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Abstract This section describes the characteristics of optical waveguides in vari- ous material systems. The main focus will be on the properties of glass fiber whose different types and production methods will be described. The material systems of optical waveguides, which are described in this chapter, are displayed in the 6 Nov 2016 The expression of the group velocity vg=???kz. For the waveguide with wave vector kz in propagation direction is correct. And the phase velocity is vph=?kz.

According to the dispersion relation of the wave guide, when? Approaches the cut-off frequency?c=c?a, the wave vector kz and and group 5 Mar 2014 Lets explain cutoff frequency, guide wavelength, group velocity, phase velocity and propagation constant of a waveguide.

Cutoff frequency: Cutoff frequency is the frequency below which attenuation occurs and above which propagation takes place.Each mode have a specific cutoff frequency. For TEmn Light, Phase and Group Velocities. Typically, coax vendors will quote a velocity factor for their wares. If the 'stuffing' in a coax cable is PTFE, with a dielectric constant (relative permittivity) of 2.07, the velocity factor is 69.5%. Velocity of light for quasi-TEM transmission lines such as microstrip and coplanar waveguide is equal Transit-time measurements of the leading edge of pulse-modulated microwaves propagating in a waveguide have been performed in time domain. These measurements show that a part of the energy associated with the leading edge of the pulse propagates with a phase velocity exceeding the intrinsic velocity in free space Waveguide. • Waveguide = hollow metal pipe.

• Rectangular, 2:1 aspect ratio. • Advantages. – Conducts millimeter signals. – High power Wave velocity: Free space propagation. • Group velocity: Energy propagation.

• Phase velocity: Phase propagation. / c f / F cutoff1. The phase velocity of a wave is the rate at which the phase of the wave propagates in space. This is the velocity at which the phase of any one frequency component of the wave travels. For such a component, any given phase of the wave (for example, the crest) will appear to travel at the phase velocity. The phase velocity Group Velocity. It is the velocity of electromagnetic waves in the waveguide.

Group velocity is denoted by the symbol Vg. Group velocity is less than velocity of electromagnetic waves in free space. Phase Velocity.

It is the velocity which the electromagnetic waves changes it phase in the waveguide during propagation. This page contains some of important equations for rectangular waveguide. Here is an index to the subject of waveguide mathematics: Cutoff frequencies. Guide wavelength. Phase velocity and group velocity. Group delay in waveguide.

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1 Gathering tradeskills. 1.1 Mining; 1.2 Relic Hunter; 1.3 Survivalist. 2 Production tradeskills. 2.1 Architect; 2.2 Armorer; 2.3 Outfitter; 2.4 Tailor; 2.5 Technologist; 2.6 Weaponsmith. 3.1 Cooking; 3.2 Farmer. 4.1 Runecrafting; 4.2 Salvaging Many trades are in high demand of metals--and so a Miner is often a good compliment to other Tradeskills such as Armorer and Weaponsmith. If you find the glint of gems and the sound of clinking metal to be enticing, see if Mining is your forte!

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